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Irbesartan: Angiotensin II is usually a strong vasoconstrictor fashioned from angiotensin I inside of a reaction catalyzed by angiotensin-changing enzyme (ACE, kininase II). Angiotensin II will be the principal pressor agent of the RAS and in addition stimulates aldosterone synthesis and secretion by adrenal cortex, cardiac contraction, renal resorption of sodium, action of your sympathetic nervous program, and sleek muscle cell development.
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Irbesartan blocks the vasoconstrictor and aldosterone-secreting effects of angiotensin II by selectively binding to the AT1 angiotensin II receptor. There may be also an AT2 receptor in many tissues, but It's not at all involved in cardiovascular homeostasis.
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Blockade of the AT1 receptor gets rid of the unfavorable responses of angiotensin II on renin secretion, although the ensuing improved plasma renin activity and circulating angiotensin II will not triumph over the consequences of irbesartan on hypertension.
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